Synergistic interactions of blood-borne immune cells, fibroblasts and extracellular matrix drive repair in an in vitro peri-implant wound healing model.

A decreasing trend of Fn and fibrin volumes ( Fig. 4g,h ), together with voids in co-culture compared to blood only conditions ( Fig. 5a ), suggest that fibroblasts actively degrade, remodel and integrate into dense blood clots.

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Significant differences between donors were found for Fn, fibrin, CD41 and Fn fraction delocalized with fibrin, still showing highly significant differences between conditions.

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