A three amino acid deletion in the transmembrane domain of the nicotinic acetylcholine receptor α6 subunit confers high-level resistance to spinosad in Plutella xylostella.

This result demonstrates that there is highly significant, tight genetic linkage between the 3-aa deletion mutation and spinosad resistance in the SZ-SpinR strain (P < 0.0001, Fisher's exact test), and confirms that the 3-aa deletion mutation is recessive. 3.4.

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