In this case, the regulation of opioid synthesis by DA, which can be measured, is highly significant. 6 , 7 The hypotheses concern three main mechanisms: endorphin deficiency, the presence of abnormal endorphins, and the excess of endorphins in schizophrenia. 8 Some authors postulate that BE may have therapeutic effects in patients with schizophrenia, 9 but their findings are contradictory.
← all excerpts
Comparison of beta-endorphin and CGRP levels before and after treatment for severe schizophrenia.
1
—
—