miR-218 Involvement in Cardiomyocyte Hypertrophy Is Likely through Targeting REST.

We further observed the effect of miR-218 on controlling the expression levels of REST by real-time PCR and Western blot, respectively, in H9C2 cells, and found that upregulation of miR-218 exhibited a decreasing trend in mRNA levels of REST ( Figure 3 C) and significantly inhibited REST expression in protein levels ( Figure 3 D).

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