Moreover, our data showing a marginally significant rescue of the monopolar phenotype by the overexpression of TUB1 and TUB2 implicates spindle breakage in causing a deficiency in the intracellular pool of assembly-competent tubulin available for progeny cells during subsequent mitoses as previously suggested [ 52 ].
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Timely Endocytosis of Cytokinetic Enzymes Prevents Premature Spindle Breakage during Mitotic Exit.
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