A new glucocerebrosidase-deficient neuronal cell model provides a tool to probe pathophysiology and therapeutics for Gaucher disease.

Dis Model Mech · 2016 · PMC4958308 · PMID 27482815

Europe PMC DOI

hedged sentence

0.0017

closest p · 0.0× alpha

0.0017

boldest claim

The sentences

highly significantP =0.0017actually significant

g ba −/− immortalized neurons showed a severe deficiency in enzyme activity (3.40±0.36% relative GCase activity, mean±s.e.m.) compared with gba +/+ immortalized neurons (93.44±12.26% relative GCase activity), which was highly significant ( P =0.0017) ( Fig. 3 C).

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