Sam68 promotes self-renewal and glycolytic metabolism in mouse neural progenitor cells by modulating <i>Aldh1a3</i> pre-mRNA 3'-end processing.

In agreement with a direct modulation of Aldh1a3 expression by Sam68, we also observed a highly significant correlation between expression of these genes in post-natal brain, with highest levels of both transcripts in olfactory bulb and cerebellum and lowest expression in the hypothalamus ( Figure 6E ).

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