A network of epigenetic modifiers and DNA repair genes controls tissue-specific copy number alteration preference.

To test whether the results of this analysis depend on the reference epigenomes that we selected, we performed this comparison for different tissue-of-origin associations ( Supplementary file 2 ) and observed a significant or marginally significant difference in length distributions in all cases (p ≤ 0.05).

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A modestly significant enrichment for H3K9me3 and depletion of DNA methylation for the copy number alteration was then reported.

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