Analysis of KT-MT attachment status in MG132-treated BubR1 Δ660–685 mutant expressing cells demonstrated a highly significant increase in laterally attached kinetochores (Fig. 4d,e ) in both congressed and uncongressed kinetochores (Supplementary Fig. 7A ).
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Aurora-B kinase pathway controls the lateral to end-on conversion of kinetochore-microtubule attachments in human cells.
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