Thus, highly significant QTL peak, Ath44 , on Chr1 at 68 cM was present only in the DBA-apoE x 129-apoE F2 population ( Fig 4A , Table 5 ).
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Genetic architecture of atherosclerosis dissected by QTL analyses in three F2 intercrosses of apolipoprotein E-null mice on C57BL6/J, DBA/2J and 129S6/SvEvTac backgrounds.
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