IL-17A Enhances Microglial Response to OGD by Regulating p53 and PI3K/Akt Pathways with Involvement of ROS/HMGB1.

Front Mol Neurosci · 2017 · PMC5583146 · PMID 28912678

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an increasing trendno p-value reported

The results demonstrated that the production of a large amount of ROS may promote IL-17A and HMGB1 expression, and the down-regulation of IL-17A or HMGB1 expression caused a reduction in the production of ROS (Figure 9D ), which, however, still appeared as an increasing trend relative to the control group.

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