Acute maternal oxidant exposure causes susceptibility of the fetal brain to inflammation and oxidative stress.

J Neuroinflammation · 2017 · PMC5622443 · PMID 28962577

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an overall trendno p-value reported

Surprisingly, none of the chemokines measured showed any significant changes in gene expression in either maternal or fetal brain; however, an overall trend of upregulation of M1-associated chemokines (CCL3, CCL4, CCL7, CCL9) and downregulation of M2-associated chemokines (CCL2, CXCL10, and CCL22) was observed in GSH depleted fetal brain.

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