This last observation is interesting because it suggests that plasma TNAP activity or TNAP vesicular shedding might be regulated by an interaction between age and diet, however this single highly significant observation might has also occurred by chance considering the large number of parameters investigated.
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Overexpression of tissue-nonspecific alkaline phosphatase (TNAP) in endothelial cells accelerates coronary artery disease in a mouse model of familial hypercholesterolemia.
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