It is possible that altered NO signaling contributes to vascular abnormalities at baseline in the R 213 G mice as our data support an overall trend towards lower levels of lung GTPCH-1 compared to WT.
← all excerpts
Redistribution of Extracellular Superoxide Dismutase Causes Neonatal Pulmonary Vascular Remodeling and PH but Protects Against Experimental Bronchopulmonary Dysplasia.
1
—
—