Mitochondrial abnormalities and disruption of the neuromuscular junction precede the clinical phenotype and motor neuron loss in hFUSWT transgenic mice.

Hum Mol Genet · 2018 · PMC5886082 · PMID 29194538

Europe PMC DOI

hedged sentence

0.0500

closest p · 1.0× alpha

0.0500

boldest claim

The sentences

highly significantP value of <0.05actually significant

A P value of <0.001 is considered to be highly significant, and P value of <0.05 significant.

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