The mycoparasitic fungus <i>Clonostachys rosea</i> responds with both common and specific gene expression during interspecific interactions with fungal prey.

This may be significant as the 50‐fold induction of chiC1 against F. graminearum , but not against B. cinerea , may suggest adaptation of killer toxin‐like paralogs towards different fungal preys (driven by differences in cell wall composition, presence of chitinase inhibitors etc.).

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