Ischemic Postconditioning Regulates the Glu Level and Immune Status in CNS and May Reduce the Risk of Poststroke Suppression The development of comorbid depression after stroke is clinically highly significant because poststroke depression is associated with increased mortality, slows recovery, and leads to worse functional outcomes.
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Cerebral Ischemic Postconditioning Plays a Neuroprotective Role through Regulation of Central and Peripheral Glutamate.
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