The defect was highly significant: MGs transfected with LentiCon showed an average length of 5.6 μm, whereas the length was reduced to 1.5 μm by RelB KD and to 0.6–0.8 μm by Tfg or BAF45d KD ( Fig. 4 E ).
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Eda-activated RelB recruits an SWI/SNF (BAF) chromatin-remodeling complex and initiates gene transcription in skin appendage formation.
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