Follistatin-Like 3 Enhances the Function of Endothelial Cells Derived from Pluripotent Stem Cells by Facilitating β-Catenin Nuclear Translocation Through Inhibition of Glycogen Synthase Kinase-3β Activity.

Indeed, the findings of the present study demonstrate an important role of FSTL3 in iPS‐ECs, and in the early stages of EC differentiation, which may be significant toward our aim of generating therapeutically relevant ECs for use in both regenerative medicine and treatment of ischemic diseases.

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