HU–DNA interactions seem to be non-specific; therefore, we hypothesized that the differences in the genomic architecture between the serovars Typhimurium and Enteritidis may be significant for the different phenotypes.
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HU-Lacking Mutants of <i>Salmonella enterica</i> Enteritidis Are Highly Attenuated and Can Induce Protection in Murine Model of Infection.
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