Our study also indicated that there was a decreasing trend of spermatogenesis ability, and an increasing rate of sperm malformation and pathological damage, in testicular tissue in male offspring exposed to 0.2 μg/mL BPA, although this was not statistically different when compared with controls.
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Decreased Capacity for Sperm Production Induced by Perinatal Bisphenol A Exposure Is Associated with an Increased Inflammatory Response in the Offspring of C57BL/6 Male Mice.
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