Neuritis and vinblastine-induced axonal transport disruption lead to signs of altered dorsal horn excitability.

Although not quite significant, there was a notable reduction in the number of action potentials evoked by Aδ- and C-fiber neurons in the vinblastine-treated group compared to neuritis (Aδ-fiber neurons: F (3, 37) = 3.17, p < 0.05; C-fiber neurons: F (3, 37) = 2.97, p < 0.05, one-way ANOVA; p = 0.06 and 0.07, respectively, Bonferroni’s post hoc tests).

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