For example, when in vivo insults are delivered to an animal (chemoconvulsant, injury, genetic lesion, and so on), there may be significant delays from the time of insult to the first spontaneous seizure.
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How do we use in vitro models to understand epileptiform and ictal activity? A report of the TASK1-WG4 group of the ILAE/AES Joint Translational Task Force.
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