Rhynchophylline myocardial ischemia reperfusion injury reactive oxygen species mitochondrial permeability transition pore caspase-3/9 mitochondria pmc-status-qastatus 0 pmc-status-live yes pmc-status-embargo no pmc-status-released yes pmc-prop-open-access yes pmc-prop-olf no pmc-prop-manuscript no pmc-prop-legally-suppressed no pmc-prop-has-pdf yes pmc-prop-has-supplement no pmc-prop-pdf-only no pmc-prop-suppress-copyright no pmc-prop-is-real-version no pmc-prop-is-scanned-article no pmc-prop-preprint no pmc-prop-in-epmc yes pmc-license-ref CC BY-NC-ND Introduction An increasing trend has been observed in the incidence of coronary atherosclerotic heart disease, which is seen as one of the most common diseases that threatens human health ( 1 ).
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Rhynchophylline ameliorates myocardial ischemia/reperfusion injury through the modulation of mitochondrial mechanisms to mediate myocardial apoptosis.
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