The second possibility is that Orai1-dependent Ca 2+ entry is not the main source of Ca 2+ influx in these cells, but other Ca 2+ channels, such as TRPC (Transient Receptor Potential channels Canonical) [ 46 ] or TRPV (Transient Receptor Potential channels Vanilloid) [ 47 ] channels, may be significant contributors to Ca 2+ entry. 4.
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Role of Calcium Signaling in GA101-Induced Cell Death in Malignant Human B Cells.
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