In addition, it is highly significant to note that xenograft tumor formation and growth, as demonstrated in tumor size and tumor weight, also showed that ectopic expression of MIR190A promoted tumor formation and growth, while knockdown of ATG7 in UMUC3(MIR190A) cells completely abolished the promotive effect of MIR190A on xenograft tumor growth (Figure 8 H–J).
← all excerpts
ATG7 Promotes Bladder Cancer Invasion via Autophagy-Mediated Increased ARHGDIB mRNA Stability.
1
—
—