Humoral response to <i>Clostridium difficile</i> in inflammatory bowel disease, including correlation with immunomodulatory treatment.

We analyzed if the different levels of IgG to toxin B between treated and untreated CD patients could be attributed to a single type of DMAID, but no statistical significance due to any of the DMAID types (listed in Table 2 ) was found (as all but five DMAID‐treated patients had azathioprine and only three had mesalazine, it was statistically meaningful only to note a slight trend ( P = 0.22) for higher IgG to toxin B in patients with TNF inhibitors compared with DMAID except TNF inhibition).

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