Thrombin-cleaved syndecan-3/-4 ectodomain fragments mediate endothelial barrier dysfunction.

PLoS One · 2019 · PMC6519803 · PMID 31091226

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highly significantno p-value reported

In the presence of the PAR1 antagonist, rhS3ED and rhS4ED fragments both continued to produce highly significant decreases in TER ( Fig 7G and 7H ), suggesting the residual amount of thrombin in the treatment had a minimal effect in the TER response to syndecan ectodomain fragments.

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