Genome-wide analysis identifies rare copy number variations associated with inflammatory bowel disease.

Furthermore, we also observed some other immune process related gene sets were marginally significant in the genes affected by rare IBD-associated CNVs with the adjusted p-value of the overrepresentation < 0.25 ( S3 Fig ).

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In turn, the GTF2F1 gene demonstrated upregulation in response to enterovirus infection.[ 46 ] The highly significant IBD associated deletion over the DUSP22 gene deserves a special interest.

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