Kaplan–Meier analyses of DFS over time showed that although expression of GIV at high levels was associated with disease progression and poorer survival in both low- and high-PDE groups, the risk of progression was not statistically significant in the high-PDE state ( Figure 5E ) but was highly significant in the low-PDE state ( Figure 5F ).
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A predictive computational model reveals that GIV/girdin serves as a tunable valve for EGFR-stimulated cyclic AMP signals.
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