Specifically, as in tumors, class 4–6 SEMAs and class 3 SEMAs were in general more abundant in T‐N and E‐N cells, respectively, with an apparent trend of incremental divergence from NR4A3 to E‐N to T‐N (Figure 3 E; see supplementary materials, Figure S2 ; Table S2 I).
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NR4A3 fusion proteins trigger an axon guidance switch that marks the difference between EWSR1 and TAF15 translocated extraskeletal myxoid chondrosarcomas.
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