The elucidation of the critical KRAS effectors mediating PDAC pathogenesis and the specific biological processes provoked by KRAS signaling remain highly significant challenges in understanding the progression of this disease and in enabling the selection of effective drug targets [ 25 , 26 , 27 ].
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The Use of Genetically Engineered Mouse Models for Studying the Function of Mutated Driver Genes in Pancreatic Cancer.
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