The <i>Wolbachia</i> cytoplasmic incompatibility enzyme CidB targets nuclear import and protamine-histone exchange factors.

We next tested whether maternal overproduction of P32 or Nap1 (also identified as a potential CidB substrates or cofactors; Table 1 ) could suppress transgenic CI and found that overexpression of P32 showed highly significant suppression relative to the GFP control, increasing egg hatch rates by ~30% ( Figure 5b ).

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