Although galectin-9 KO tissues exhibited slightly lower expression levels of Smad2/3 and ET-1, the differences were not significantly significant between the two groups (Fig. 2 e).
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Amelioration of bleomycin-induced pulmonary fibrosis via TGF-β-induced Smad and non-Smad signaling pathways in galectin-9-deficient mice and fibroblast cells.
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