As expected, the diabetic phenotype led to a highly significant glomerular hypertrophy of both aged diabetic phenotypes when compared to the WT mice (Ins2±, A1AR-/-: 4.65±1.2 mm 2 vs.
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Podocyte Density and Albuminuria in Aging Diabetic Ins2± Mice with or Without Adenosine A1 Receptor Signaling.
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