Aspirin Improves Nonalcoholic Fatty Liver Disease and Atherosclerosis through Regulation of the PPAR<i>δ</i>-AMPK-PGC-1<i>α</i> Pathway in Dyslipidemic Conditions.

Aspirin Administration Decreased CCR2 Levels in the Spleen but Increased Mannose Receptor Levels in the Rabbits Fed a Cholesterol Diet ATP concentration in the aorta showed a decreasing trend in the rabbits fed a cholesterol diet; however, the concentration showed an increasing trend in the rabbits fed a cholesterol diet with aspirin ( Figure 6(a) ).

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