Proteasomal-Mediated Degradation of AKAP150 Accompanies AMPAR Endocytosis during cLTD.

eNeuro · 2020 · PMC7163082 · PMID 32205379

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may not be significantno p-value reported

Although it may not be significant, from the images it seems that there may be more AKAP150 concentrated at the plasma membrane after cLTP induction, with bigger and more intense clusters than in control conditions in agreement with the notion that LTP recruits more AKAP150 to the plasma membrane.

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