Enhanced accumulation of N-terminally truncated Aβ with and without pyroglutamate-11 modification in parvalbumin-expressing GABAergic neurons in idiopathic and dup15q11.2-q13 autism.

This modification of microglia function may be significant in the context of autism pathogenesis, in which prenatal and early postnatal infections have been postulated as triggering factors for development of autism [ 28 , 42 ].

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