Our data demonstrate a highly significant increase in upd2 and upd3 transcripts when IIS was activated as a result of PTEN suppression, indicating that IIS can activate JAK–STAT signaling in the gut.
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Insulin Signaling in Intestinal Stem and Progenitor Cells as an Important Determinant of Physiological and Metabolic Traits in <i>Drosophila</i>.
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