2 , 6 The fact that the COVID-19 cohort did not have a highly significant cardiovascular risk profile compared with the non-COVID-19 cohort, and the notable finding of significantly more frequent simultaneous thrombosis support the hypothesis of a systemic prothrombotic state associated with SARS-CoV-2. 6 A higher risk of arterial thrombosis has been previously described in association with bacteremia and other respiratory viruses, 4 but the specific pathophysiology of COVID-19 disease remains an open field for basic and clinical research.
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Arterial thrombotic complications in hospitalized patients with COVID-19.
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