In contrast, untreated and flavo-treated FANCD2 − / − cells in ana-/telophase presented γH2AX foci, with the latter showing a highly significant increase in γH2AX foci compared to untreated ana-/telophase FANCD2 − / − (Figure 7C , D).
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A distinct role for recombination repair factors in an early cellular response to transcription-replication conflicts.
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