As regards the potential pathogenetic mechanisms linking AAF and low temperature, it seems conceivable that the seasonal incidence (e.g., including acute-onset as well as the reacutization form of AF, both symptomatic and silent) may be at least partially attributed to diseases with a similar seasonal pattern, for example those co-morbidities that prevail during the winter which may be significant triggers of increased sympathetic activity and cardiac arrhythmias, such as pneumonia, exacerbations of COPD, stroke and myocardial infarction.
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Incidence of acute-onset atrial fibrillation correlates with air temperature. Results of a nine-year survey.
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