Because prostatic collagens accumulate with age, and because we did not observe a net increase in overall quantity of prostatic collagen producing cells, we consider the aging-related transition in the identity of collagen producing cells to be of probable significance with respect to prostatic collagen homeostasis.
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An immunohistochemical prostate cell identification key indicates that aging shifts procollagen 1A1 production from myofibroblasts to fibroblasts in dogs prone to prostate-related urinary dysfunction.
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