Modulating redox homeostasis and cellular reprogramming through inhibited methylenetetrahydrofolate dehydrogenase 2 enzymatic activities in lung cancer.

These outcomes were further corroborated using Oncomine TM Database in three previous studies [ 19 – 21 ], showing highly significant expression (over 2-fold) of MTHFD2 in LCa compared to normal counterparts ( Figure 1D ).

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