Comparing cell-free circulating tumor DNA mutational profiles of disease-free and nonresponders patients with oropharyngeal squamous cell carcinoma.

Furthermore, the presence of the truncating mutation TP53 G325fs (27/0.62), which dysregulates p53 functionality to promote tumorigenesis instead of halting it, in the nonresponder groups implicates the consequence of a tumorigenic mutant p53 may be significant in the nonresponder groups in comparison to the responder group.

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