hHSPB1-overexpressing mice showed enhanced expression of pro-inflammatory cytokines Tnf and Il1b as well as increased immunoreactivity of astrocyte and microglial activation markers in response to ethanol-induced acute brain injury, whereas they exhibited a nonsignificant trend of decreased apoptosis.
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Neuroinflammatory processes are augmented in mice overexpressing human heat-shock protein B1 following ethanol-induced brain injury.
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