In CSF, but not in the circulation, repeated intrathecal application of TCA evoked increased concentrations of IGFBP-3 and with borderline significance also of IGFBP-2.
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Reduced Fragmentation of IGFBP-2 and IGFBP-3 as a Potential Mechanism for Decreased Ratio of IGF-II to IGFBPs in Cerebrospinal Fluid in Response to Repeated Intrathecal Administration of Triamcinolone Acetonide in Patients With Multiple Sclerosis.
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