After operation, the ability of antioxidative stress of the skin flap decreased and showed an obvious trend of promoting apoptosis. 3.2.
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Apelin/APJ-Manipulated CaMKK/AMPK/GSK3<i>β</i> Signaling Works as an Endogenous Counterinjury Mechanism in Promoting the Vitality of Random-Pattern Skin Flaps.
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