We observed a decreasing trend for ANPODRT expression during IDD pathogenesis, and the well-recognized oxidative stress inducer TBHP downregulated ANPODRT in human NP cells in a dose- and time-dependent manner.
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Long Noncoding RNA ANPODRT Overexpression Protects Nucleus Pulposus Cells from Oxidative Stress and Apoptosis by Activating Keap1-Nrf2 Signaling.
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