This suggests that pulmonary vascular dilatations (or intrapulmonary arterial–venous anastomoses) are not a major mechanism of hypoxemia in classical ARDS but may be significant contributors to hypoxemia in COVID-19 respiratory failure.
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This suggests that pulmonary vascular dilatations (or intrapulmonary arterial–venous anastomoses) are not a major mechanism of hypoxemia in classical ARDS but may be significant contributors to hypoxemia in COVID-19 respiratory failure.